What type of glomerulonephritis is associated with IgA nephropathy?

IgA nephropathy, also known as Bergers disease, is characterized by the deposition of immunoglobulin A (IgA) in the mesangial deposits of the glomeruli. The condition is often associated with specific clinical scenarios, particularly those where symptoms appear after infections, such as after a respiratory or gastrointestinal infection. This temporal correlation is indicative of what is termed synpharyngitic glomerulonephritis, meaning that the kidney inflammation coincides with an upper respiratory tract infection, leading to the manifestation of hematuria and proteinuria.

While other forms of glomerulonephritis, such as postinfectious or those leading to nephrotic syndrome, exist, they do not fit the specific association with IgA nephropathy as closely as synpharyngitic glomerulonephritis. For example, postinfectious glomerulonephritis typically occurs after infections caused by certain organisms, often leading to a different pattern of presentation and pathophysiology compared to IgA nephropathy. Similarly, nephrotic syndrome and minimal change disease involve different mechanisms of kidney damage and proteinuria that do not directly relate to IgA deposition in the glomeruli.

By recognizing the connection between IgA